Orthopedics

Hill-Sachs Lesion: Definition, Causes, Diagnosis, and Treatment

By Jordan 7 min read

A Hill-Sachs lesion is an indentation fracture on the posterolateral aspect of the humeral head, caused when it impacts the anterior glenoid rim during an anterior shoulder dislocation.

What is the definition of Hill Sachs lesion?

A Hill-Sachs lesion is an indentation fracture, or divot, on the posterolateral (back and outer) aspect of the humeral head, which occurs when the soft bone of the humeral head impacts against the hard anterior rim of the glenoid (shoulder socket) during an anterior shoulder dislocation.

Understanding Shoulder Anatomy and Dislocation

To comprehend a Hill-Sachs lesion, it's essential to first understand the anatomy of the shoulder joint. The shoulder is a ball-and-socket joint, specifically the glenohumeral joint, formed by the head of the humerus (upper arm bone) and the glenoid fossa (a shallow socket on the scapula, or shoulder blade). This design allows for an impressive range of motion, making it the most mobile joint in the body, but also inherently less stable. Stability is provided by a complex network of ligaments, the joint capsule, and the rotator cuff muscles.

An anterior shoulder dislocation is the most common type of shoulder dislocation, where the humeral head is forced out of the glenoid socket in a forward direction. This typically results from a traumatic event, such as a fall onto an outstretched arm, a direct blow to the shoulder, or an abduction and external rotation injury.

Mechanism of Injury: How a Hill-Sachs Lesion Forms

A Hill-Sachs lesion is a direct consequence of an anterior shoulder dislocation. As the humeral head dislocates anteriorly, it is violently driven against the unyielding, bony anterior rim of the glenoid. The relatively softer bone of the posterior-lateral aspect of the humeral head then sustains a compression or impaction fracture, creating a characteristic divot or defect.

  • Impact Point: The lesion always occurs on the posterolateral aspect of the humeral head because this is the area that makes contact with the anterior glenoid rim during the dislocation.
  • Bone Density: The contrast in bone density between the humeral head and the glenoid rim facilitates this impaction injury.
  • Recurrence: The presence of a Hill-Sachs lesion is a significant risk factor for recurrent shoulder instability and subsequent dislocations.

Clinical Significance and Associated Conditions

The presence and size of a Hill-Sachs lesion are crucial for determining the prognosis and treatment strategy for shoulder instability.

  • Recurrent Dislocation: A larger or "engaging" Hill-Sachs lesion (where the defect can engage or "catch" on the anterior glenoid rim during certain arm movements) can act like a wedge, facilitating further dislocations even with less force. This creates a vicious cycle of instability.
  • Engaging vs. Non-Engaging:
    • Non-Engaging Lesion: The lesion does not consistently contact the glenoid rim during normal shoulder motion or even during provocative movements. These are less likely to contribute to recurrent instability.
    • Engaging Lesion: The lesion consistently contacts and "engages" with the glenoid rim, significantly increasing the risk of re-dislocation.
  • Associated Injuries: Hill-Sachs lesions frequently occur alongside other shoulder injuries, most notably:
    • Bankart Lesion: A tear of the anterior glenoid labrum, often accompanied by a fracture of the anterior glenoid bone (bony Bankart lesion). This is the result of the same traumatic event that causes the Hill-Sachs lesion.
    • Rotator Cuff Tears: Especially in older individuals.
    • Capsular Laxity: Stretching or tearing of the joint capsule.

Diagnosis of a Hill-Sachs Lesion

Diagnosing a Hill-Sachs lesion typically involves a combination of clinical assessment and imaging.

  • Clinical History: A history of one or more anterior shoulder dislocations is the primary indicator.
  • Physical Examination: Assessment of shoulder stability, range of motion, and specific tests for instability.
  • Imaging Studies:
    • X-rays: Standard anterior-posterior (AP) views may not always clearly show a Hill-Sachs lesion. Specialized views, such as the Stryker notch view or West Point axillary view, are often required to visualize the posterolateral humeral head.
    • Magnetic Resonance Imaging (MRI): Considered the gold standard for soft tissue and bone pathology. An MRI can accurately visualize the size and depth of the Hill-Sachs lesion, as well as identify concomitant injuries like Bankart lesions or rotator cuff tears.
    • Computed Tomography (CT) Scan: Particularly useful for assessing the bone defect's size, depth, and whether it's an engaging lesion, especially when planning surgical intervention.

Treatment Considerations

Treatment for a Hill-Sachs lesion depends heavily on its size, whether it's engaging, the patient's activity level, and the presence of recurrent instability.

  • Conservative Management: For small, non-engaging lesions in patients with minimal instability, non-surgical approaches may be considered. This typically involves:
    • Rehabilitation: Focusing on strengthening the rotator cuff and scapular stabilizing muscles, restoring proprioception, and improving neuromuscular control.
    • Activity Modification: Avoiding positions that provoke instability.
  • Surgical Intervention: Surgery is often recommended for larger, engaging lesions, recurrent instability, or in high-demand athletes. Common surgical procedures include:
    • Arthroscopic Capsulolabral Repair (Bankart Repair): If a Bankart lesion is present, this procedure reattaches the torn labrum to the glenoid, improving stability.
    • Remplissage Procedure: An arthroscopic technique that involves filling the Hill-Sachs defect by suturing the posterior joint capsule and infraspinatus tendon into the lesion. This effectively converts an "engaging" lesion into a "non-engaging" one by obliterating the defect.
    • Bone Augmentation: For very large Hill-Sachs lesions, bone grafts (autograft or allograft) may be used to fill the defect and restore the normal contour of the humeral head.
    • Latarjet Procedure: While primarily addressing glenoid bone loss, this procedure (transferring a piece of the coracoid process with its attached muscles to the anterior glenoid) is sometimes performed in cases of significant glenoid bone loss that often co-occurs with large Hill-Sachs lesions.

Rehabilitation and Long-Term Management

Post-surgical rehabilitation is critical for optimal outcomes, regardless of the procedure performed.

  • Phased Approach: Rehab progresses through phases, starting with protecting the repair (immobilization), followed by restoring range of motion, gradually increasing strength, and finally returning to sport-specific activities.
  • Focus Areas: Emphasis is placed on strengthening the rotator cuff, scapular stabilizers, and deltoid muscles, along with proprioceptive training to re-educate the joint's sense of position and movement.
  • Adherence: Patient adherence to the rehabilitation protocol is paramount to prevent re-injury and achieve full functional recovery.

Prevention

While not all shoulder dislocations can be prevented, especially in traumatic accidents, certain measures can reduce the risk of initial and recurrent dislocations:

  • Proper Management of Initial Dislocation: Timely and correct reduction of the initial dislocation, followed by appropriate immobilization and rehabilitation.
  • Strength Training: Developing strong rotator cuff and scapular stabilizing muscles to enhance dynamic shoulder stability.
  • Proprioceptive Training: Exercises that improve joint awareness and control.
  • Sport-Specific Training: Incorporating exercises that mimic movements in sports to build resilience and prepare the shoulder for specific demands.

Conclusion

A Hill-Sachs lesion is a distinct bony injury on the humeral head resulting from an anterior shoulder dislocation. Its presence and characteristics significantly influence the risk of recurrent instability and guide treatment decisions. A comprehensive understanding of this lesion, from its biomechanics to its management, is vital for clinicians and individuals dealing with shoulder instability to achieve successful recovery and prevent future complications.

Key Takeaways

  • A Hill-Sachs lesion is an impaction fracture on the humeral head, specifically caused by an anterior shoulder dislocation.
  • The lesion forms when the softer bone of the humeral head collides with the harder anterior rim of the glenoid socket.
  • Its presence, especially if it's an "engaging" lesion, significantly increases the risk of recurrent shoulder instability and re-dislocations.
  • Diagnosis involves clinical assessment and imaging studies like X-rays (specialized views), MRI, and CT scans.
  • Treatment ranges from conservative management for small, non-engaging lesions to surgical intervention for larger or recurrent instability, followed by crucial rehabilitation.

Frequently Asked Questions

What causes a Hill-Sachs lesion?

A Hill-Sachs lesion is caused by an anterior shoulder dislocation, where the humeral head impacts and creates a divot on its posterolateral aspect against the anterior rim of the glenoid.

Why is a Hill-Sachs lesion important?

The presence and size of a Hill-Sachs lesion are crucial because it can significantly increase the risk of recurrent shoulder instability and subsequent dislocations, especially if it's an "engaging" lesion.

How is a Hill-Sachs lesion diagnosed?

Diagnosis typically involves a clinical history of shoulder dislocations, physical examination, and imaging studies such as specialized X-rays, MRI, and CT scans, with MRI being the gold standard for soft tissue and bone pathology.

Can a Hill-Sachs lesion be treated without surgery?

Conservative management with rehabilitation and activity modification may be considered for small, non-engaging lesions in patients with minimal instability, focusing on strengthening and neuromuscular control.

What are common surgical treatments for a Hill-Sachs lesion?

Surgical options for Hill-Sachs lesions include arthroscopic capsulolabral repair (Bankart repair), the Remplissage procedure to fill the defect, bone augmentation for very large lesions, or the Latarjet procedure, often followed by extensive rehabilitation.