Musculoskeletal Health

Hypermobile Body Type: Understanding Joint Hypermobility, Causes, and Assessment

By Jordan 4 min read

A hypermobile body type is characterized by joints with an unusually large range of motion, often due to genetic factors affecting connective tissues, and exists on a spectrum from asymptomatic flexibility to symptomatic instability.

What is a hypermobile body type?

A hypermobile body type is characterized by joints that have an unusually large range of motion, extending beyond the typical physiological limits. While often associated with enhanced flexibility, it exists on a spectrum from asymptomatic bendiness to symptomatic joint instability and chronic pain, sometimes indicative of underlying connective tissue disorders.

Understanding Joint Hypermobility

Joint hypermobility refers to the ability of a joint to move beyond its normal anatomical range. This excessive laxity can manifest in various ways, from a single "bendy" joint (localized hypermobility) to multiple joints across the body (generalized hypermobility). It's crucial to distinguish between natural flexibility, which is beneficial, and hypermobility, which implies a structural difference that allows for greater, and sometimes uncontrolled, movement.

Normal vs. Excessive Range of Motion (ROM): Every joint has a normal ROM determined by its structure (bone shape, ligaments, joint capsule, muscles). In a hypermobile individual, the passive structures (ligaments and joint capsule) that typically limit movement are more extensible, allowing for greater excursion.

Causes and Contributing Factors

The primary cause of joint hypermobility is often genetic, affecting the composition and structure of connective tissues.

  • Genetic Predisposition: Many forms of hypermobility are hereditary, passed down through families. This often involves variations in the genes responsible for producing collagen.
  • Collagen Structure: Collagen is the most abundant protein in the body, providing strength and elasticity to connective tissues like ligaments, tendons, skin, and joint capsules. In hypermobile individuals, the collagen may be more pliable or "stretchy" than usual. This is a hallmark of conditions such as Hypermobility Spectrum Disorder (HSD) and Hypermobile Ehlers-Danlos Syndrome (hEDS), where the connective tissue is inherently weaker or more elastic.
  • Muscle Tone: Some individuals with hypermobility may also have lower resting muscle tone, which can contribute to reduced joint stability.
  • Bone Structure: Shallow joint sockets (e.g., hip or shoulder) can allow for greater movement and predispose individuals to hypermobility.

Characteristics and Assessment

Identifying a hypermobile body type often involves observing specific physical signs and, in clinical settings, using standardized assessment tools.

  • Common Signs of Hypermobility:
    • Hyperextension: Elbows or knees extending beyond 10 degrees past straight.
    • Thumb to Forearm: Ability to bend the thumb back to touch the forearm.
    • Finger Extension: Ability to extend the pinky finger back beyond 90 degrees.
    • Palm to Floor: Ability to place palms flat on the floor with knees straight during a forward bend.
    • Shoulder/Hip Flexibility: Unusual range of motion in these large joints.
  • The Beighton Score: This is a widely used 9-point system to assess generalized joint hypermobility. It involves five maneuvers, each assessed bilaterally (except for the spine flexion):
    1. Passive dorsiflexion of the 5th metacarpophalangeal joint to 90 degrees or more (1 point per hand).
    2. Passive apposition of the thumb to the forearm (1 point per hand).
    3. Hyperextension of the elbow beyond 10 degrees (1 point per arm).
    4. Hyperextension of the knee beyond 10 degrees (1 point per leg).
    5. Forward flexion of the trunk with knees straight and palms flat on the floor (1 point). A score of 4/9 or more is typically indicative of generalized joint hypermobility, though thresholds can vary.
  • Distinguishing Asymptomatic Hypermobility from Symptomatic Conditions: It's vital to note that many people with hypermobility experience

Key Takeaways

  • Hypermobility involves joints moving beyond their typical range, differing from beneficial flexibility, and can be localized or generalized.
  • It's primarily genetic, often linked to variations in collagen, which makes connective tissues more pliable and affects ligaments, tendons, and joint capsules.
  • Common signs include hyperextending elbows/knees, thumb-to-forearm touch, and high scores on the Beighton system, a 9-point assessment tool.
  • It's crucial to distinguish between asymptomatic hypermobility and symptomatic conditions like Hypermobility Spectrum Disorder (HSD) or Hypermobile Ehlers-Danlos Syndrome (hEDS).

Frequently Asked Questions

What defines a hypermobile body type?

A hypermobile body type is characterized by joints that have an unusually large range of motion, extending beyond typical physiological limits, due to more extensible ligaments and joint capsules.

What are the main causes of joint hypermobility?

The primary cause of joint hypermobility is often genetic, involving variations in collagen structure, which makes connective tissues like ligaments and tendons more pliable or "stretchy."

How is generalized joint hypermobility assessed?

Generalized joint hypermobility is commonly assessed using the Beighton Score, a 9-point system evaluating specific joint movements like elbow/knee hyperextension and thumb-to-forearm apposition.

Is all flexibility considered hypermobility?

No, it's crucial to distinguish between natural flexibility, which is beneficial, and hypermobility, which implies a structural difference allowing for greater, and sometimes uncontrolled, movement.

What role does collagen play in hypermobility?

Collagen, a protein providing strength and elasticity to connective tissues, may be more pliable or "stretchy" in hypermobile individuals due to genetic variations, leading to increased joint laxity.